Anti-aging food

It is important to understand that a number of vitamin work more effectively in the body by the presence of antioxidants. When consumed anti-oxidants fight will stick with him on the free radical, and ultimately reduce the inflammation associated with many aspects of the aging process. Not limited to heart disease and wrinkling only.

From a number of nutritious foods, there are three foods that should be considered eligible to slow aging, namely salmon, bluberi, and green tea.

Salmon contain antioxidants that make a very good vitamins C and E work longer, improve the health of the eyes and skin. Salmon is also a source of protein needed to create all the cells, skin, hair, eyes, muscles, and organs. Also contain healthy fats, the Omega-3, which increase the HDL to reduce the negative effects of LDL cholesterol.

Give antosianin that help contain vitamin C and E work better in the body. Research conducted in Denmark shows that give used in the reduction of inflammation and treatment or prevention of Alzheimer's Disease, Parkinson, senile, and mood disturbance or mood.

Green tea is healthful beverage. Month last February, written in the journal of Nutrition and Biochem, by the Japanese researchers, katekin in the green tea is a source of antioxidants guard for the brain neurons and prevent the decline in cognitive.

Three strong antioksidatif food should be included in the menu of food daily to maintain mental and physical health. In addition, also added fruits and vegetables rich in antioxidants, such as:

• avocado. Fruit is a good source for saturated fat is not a single can help lower the rate of bad cholesterol in the body. Avocado also become somber vitamin E can help maintain health and prevent skin aging skin. Vitamin E may also help simtom hot flush or burning feeling in the face. Rich, potassium, which will help prevent fluid containment and high blood pressure.

• Give. Give black, bluberi, black wine contains fitokimia known as a strong flavonoid antioxidants, which help protect the body against damage caused by free radicals and aging.

• Cabbage. Cruciferous family of vegetables, including cabbage, cabbage flowers, broccoli, Brussels sprout, radish, help the body fight the toxins and cancer. Suggested consume about 115 grams of vegetables each day. When possible, the consumption of raw or boiled so that when the enzyme is important remain intact.

• Garlic. Protective effect against heart of garlic have been recorded since long. Results of research in 1994, in Iowa, U.S., against 41,837 women aged 55-69 years shows that women who consume one cloves garlic at least once each week 50 percent lower risk of colon cancer development. Research in other Tasgore Medical College, India, shows that garlic reduce the rate of cholesterol and help thin the blood instead of aspirin is more effective and also reduce the risk of heart disease.

• Ginger. Berempah the root of this system can improve digestion and circulation in the body that is very useful, especially for the elderly. Ginger also can help ease pain and rheumatism pains.

• Nuts. Nuts are good sources of minerals, particularly brazil nuts and the walnut tree. Although berkalori high, canary-rich potassium, magnesium, iron, ZINC, copper and selenium. Add the beans in your diet can improve the function of channel digested and immune system, improve skin and prevent cancer. Nuts also help control the rate of cholesterol.

• beer, wine, yoghurt. Rich will be important minerals such as potassium, calcium, protein, and vitamin B. Another thing that makes beer, wine, yoghurt become one of the many important food is the presence of bacteria living in it. These bacteria help the absorption of the vitamin in the intestine and stabilize the body's immunity system.

• Rice and paste red wheat. Carbohydrate is the food berenergi. Replace the pasta and white rice pasta with red wheat and rice, and you will quickly feel the difference in the rate of energy.

• watermelon. Not only have the effect of alkali (a basa) to the body, but also provide essential liquids that the body needed to perform various tasks.

• Water. No one can match the intensity of water. Removing the poison from the water body also provides for fluid flow of blood.

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Vitamins and Minerals to Prevent Depression

Food that is rich in the fatty acid Omega-3, such as nuts and fish oils such as salmon, tuna, Trout, and Mackerel, also considered helpful for reducing depression. Dr. Research. Andrew Stoll in 1999 shows that depression patients who consume fatty acid Omega-3-regularly will have a level of depression, lower.

In research published in November reported that 20,022 patients depression that is not susceptible to drugs, can be reduced symptoms after obtaining Feed Omega-3. Joseph Hibbeln, a senior medical researchers, found that residents of Finland who consume fish at least twice a week to decrease the risk of depression, 50 percent.

Eating rice can also reduce depression. Carbohydrate trigger out hormone insulin to the bloodstream. Hormones that clean blood from amino acids, except triptofan which is the embryo serotonin.

Serotonin is a neurotransmitter that is able to reduce pain, control the taste of food, and provide a sense of calm. Serotonin, there are many in the chocolate, so for people with depression, chocolate is a good choice.

Along with the increased demand, especially for those living in big cities, the number of depression tend to increase every year. Of course we still remember the grievous incident in the city of Leicester, when a mother kills her children are still small with poison and eventually suicide due to depression because of the economic crush.

Risk factors that can encourage someone to do suicide soul interference is, the economy, education, health, the poor, events that cause stress, and depression.

Depression as a suicide risk factors should also be skeptical. Many of pressure in this life, can become the trigger depression. Dr. Suryo Dharmono, Sp.KJ, from the Department of psychiatry FKUI, had explained that depression is often used broadly to describe the atmosphere completely difficult, protracted suffering, life crisis, a decline in social situations and various other nuances glum.

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Thyroid Problems Potentially Linked to Glaucoma Risk

People with a thyroid disorder run an increased risk of developing the eye disease glaucoma, a new study suggests.

In fact, those with glaucoma are 38 percent more likely to have had a thyroid condition at some point in their life, said the study authors, from the University of Alabama at Birmingham. Glaucoma is the leading cause of irreversible blindness worldwide.

“Studies like this are very useful in understanding what causes this disease,” said lead researcher Gerald McGwin, vice chairman of the Department of Ophthalmology at the university’s School of Medicine.

“If we can determine that thyroid problems are related to glaucoma, then we can make some hypotheses about what the mechanism behind that relationship might be and help us understand what might be the cause of glaucoma,” McGwin said. “And that may lead to more effective treatments or preventive measures.”
The findings were published online Oct. 16 in the British Journal of Ophthalmology.

About the Study
For the study, McGwin’s team collected data on 12,376 people who participated in the 2002 National Health Interview Survey. They were asked if they’d ever been diagnosed with a thyroid problem or glaucoma. Slightly more than 4.5 percent said they had glaucoma, and 12 percent said they had been diagnosed with a thyroid problem.

Among people who had glaucoma, 6.5 percent said they had a thyroid problem, while 4.4 percent said they’d never had a thyroid condition.
People with thyroid problems should see an ophthalmologist or make their ophthalmologist aware of their thyroid condition, McGwin advised. “Somebody who has a history of thyroid problems and has not seen an ophthalmologist may have a heightened level of concern about their eyesight,” he said.

The thyroid produces hormones essential for the functioning of every cell in the body; these hormones help regulate growth and chemical reactions.
In glaucoma, the optic nerve becomes progressively damaged and, if not treated, leads to loss of vision and even blindness.

The study authors suggested that the link between glaucoma and thyroid disorders may owe to chemical deposits in the blood vessels that circulate blood to the eye, causing an increase in pressure within the eyeball. Increased pressure in the eyeball is the main feature of glaucoma.


“The Best Advice is to Get your Eyes Checked”
Dr. Andrew Iwach, a spokesman for the American Academy of Ophthalmology and executive director of the Glaucoma Center of San Francisco, said the potential link between thyroid problems and glaucoma is interesting and should be taken into account, but it still needs to be proven. The best advice is to get your eyes checked, he said.

“If you haven’t seen an ophthalmologist by age 40, that’s a great time to get a baseline exam,” Iwach said.

“People may not know they are at risk for glaucoma. You are functioning fine, and yet slowly, this disease can chip away at the optic nerve, and by the time you have symptoms from glaucoma, oftentimes there’s not really much we can do,” he said.

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Will Glaucoma be the First Neurodegenerative Disease to be Cured?

“Catalyst For a Cure” Investigators to Present their Hypotheses at GRF’s January 28 Benefit

Tuesday, November 18, 2008 — “More than in any preceding year,” says Glaucoma Research Foundation (GRF) President and CEO Thomas M. Brunner, “there is a heightened sense of anticipation around the annual Research Overview by the four principal investigators of the GRF-funded Catalyst For a Cure (CFC) research consortium that opens GRF’s 31st Anniversary Benefit and Celebration at 5 pm, Wednesday, January 28, 2009, in San Francisco’s legendary Palace Hotel.


“As the conventional understanding of glaucoma evolves from being described as an eye disease to a neurodegenerative disease,” Brunner reports, “there is an emerging sense that glaucoma could actually be the first neurodegenerative disease we can cure. And that extraordinary possibility will be an overriding theme of all the presentations, I’m sure.”

The Research Overview is co-chaired by noted Silicon Valley philanthropists and entrepreneurs, Nobuko Saito Cleary and Gary Cleary PharmD, PhD. The Overview is followed at 6 pm by The President’s Reception and presentation of The President’s Award to Rohit Varma, MD, (Doheny Eye Center, University of Southern California), for his definitive studies revealing Latino community at highest risk for glaucoma.

GRF’s highest honor, The Catalyst Award, will be presented to American Academy of Ophthalmology Executive Vice President H. Dunbar Hoskins Jr, MD, at the dinner following at 7 pm. Presenting The Catalyst Award will be the 31st Anniversary Celebration national chair, William Tasman, MD (Wilmer Eye Institute, University of Pennsylvania).

Catalyst For a Cure: A new model for scientific research

Established by GRF in 2000, the CFC was first proposed as a partnership by the Steven and Michele Kirsch Foundation on the premise that the search for a cure should be a priority, and that the pace of discovery could be accelerated with important innovations: instead of the traditional academic approach of sole scientists working in isolation, the project would bring together investigators with complementary skills working together in a genuine, real time collaboration, and recognizing that traditional ophthalmic approaches to glaucoma research were well served, we would recruit for the research team, instead, from the burgeoning world of neuroscience and genetics.

CFC Principal investigators include: David Calkins PhD, Vanderbilt University; Philip Horner PhD, University of Washington; Nicholas Marsh-Armstrong PhD, Johns Hopkins University; and Monica Vetter PhD, University of Utah. Now in its third and final three-year cycle, major funding for CFC is provided by a grant to GRF from the (insert Barr language here).

“Our CFC’s research breakthroughs from the year just ending continue to make strides both in understanding what causes glaucoma, and in conducting studies that translate the basic science into potential glaucoma treatments,” Brunner said. He cited:
Two interventional studies that firmly establish that oxidative stress is a factor in the rate of loss of cellular function from glaucoma — effectively preventing vision loss in a relevant model of glaucoma. Results of these studies were published this year in the scientific journal Investigative Ophthalmology and Visual Science and reported at the annual meeting of the Association for Research in Vision and Ophthalmology.
In another important pair of studies, the CFC discovered that the cell death that causes vision loss in glaucoma has two distinct phases, and that axonal degeneration precedes neuronal loss. These facts point to a therapeutic window for interventions. Importantly, the studies have determined that vision loss from glaucoma can be predicted by observing changes specific to genes and proteins. The CFC published both of these findings in the highly regarded Journal of Neuroscience.

Glaucoma research in broader national context

“This year we also benefited from two major activities that give us a much broader national context in which to evaluate recent findings as well as set CFC goals for the year ahead.”

The first event was a major summit meeting of internationally renowned scientists and physicians recently convened with GRF funding by David Calkins PhD at Vanderbilt University. “Unlike the current indirect glaucoma treatments that work to lower the pressure within the eye,” Dr. Calkins said, “the goal of this meeting was to discover ways to cure the disease by stopping and possibly reversing damage to the optic nerve itself. Of particular interest are the similarities between glaucoma and other chronic neurodegenerative diseases such as Alzheimer’s, Parkinson’s, Huntington’s and Lou Gehrig’s disease. The hope is that by studying the mechanisms of neurodegeneration, new targets for novel therapeutic interventions before the critical point of irreversible damage occurs.”

Second, GRF’s new Director of Scientific Programs and Licensing, Allen Poirson PhD, completed GRF’s first Glaucoma Research Status (GRS) survey, evaluating current research areas of interest, as well as funding priorities from national pace setters. “Finding a cure or therapies that can avert this threat,” reports Dr. Poirson, “is simply not an urgent national imperative. We looked at several basic indicators and documented in the government sector the low priority standing for funding new glaucoma research at the National Eye Institute and the resulting small amount of new research this low standing makes possible. In the corporate sector, there are no new ‘Mechanisms of Action’ medications in Phase 3 trials.” In fact, said Poirson, the handful of foundations like GRF in the non-profit sector funding glaucoma research provided approximately the same amount of funding for new research programs utilizing molecular and cellular biology techniques that emphasize neurodegeneration and neuroprotection pathways as did the NEI in the last two years: just $4 million!
About the Glaucoma Research Foundation

Located in San Francisco and now celebrating its 31st Anniversary year, GRF is the nation’s most experienced foundation dedicated solely to glaucoma research and education. In addition to funding innovative research like the Catalyst For a Cure research consortium and its Shaffer Grants for Innovative Glaucoma Research, GRF also is the “go to” agency for education materials, including the definitive reference for newly diagnosed, Understanding and Living with Glaucoma (available in both English and Spanish editions); a special brochure serving those at highest risk, including African-Americans, Latinos, and children; and a toll free phone line, 800-826-6693, staffed during office hours with an information specialist to handle a variety of inquiries.

For more information about GRF, call 800.826.6693. For information about tickets for the 31st Anniversary Benefit and Celebration, contact Carmen Torres.

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